Seven unique variants of coronavirus are found in the United States

According to a recent study, a team of researchers discovered seven distinctive variants of the new coronavirus, spread across the United States, new strains unique to and mutated within the US. The seven mutations of SARS-CoV-2, the virus that causes COVID-19, were discovered between August and November 2020. They were all found to be within the same growing line. The more contagious variants emerging from the UK and South Africa, according to the authors, said that seven mutant descendants in the US had no signatures of increased transmissibility and noted that none of them became prominent in the United States. “Therefore, SARS-CoV-2 variants may emerge over time and increasingly increase in the absence of any clear or sustained selective benefit.” One of the co-authors of the study, Jeremy Kamil, a virologist at Louisiana State University (LSU), spoke with The New York Times and said he was sequencing Louisiana coronavirus test samples when he came across new variants of the virus. Kamil loaded the viral strains he uncovered into an online database. After receiving an email from a researcher at the University of New Mexico, co-author of the study Daryl Domman, the two found that the same sex also appeared in both places and What is difficult about the mutation is that it occurs in a gene that affects how the virus interacts with human cells, making it possible for the virus to enter our cells effortlessly and cling to our immune system, the report said. “There is clearly something going on with this mutation,” Kamil said Times. “I think there is a clear signature of an evolutionary advantage,” he said, noting the strengths of the mutation.

The lineage discovered by Kamil found a mutation in the 677th amino acid in the genetic order, just like the other six cases across the country. The perfect superpower for a virus is the increased ability to attach to an organism more easily and effectively. To survive during the many months it has, the coronavirus has adapted to its human host in more ways than one, as scientists have proven. The variation in the 677th amino acid found in the seven mutant lines made it possible for the “peak” of the coronavirus – the part used by the virus to cling to the surface of human cells and this its characteristic form – to be activated, which can lead to a subsequent infection. This does not mean that the virus is more infected, more deadly or spreads at a faster rate, but that does mean that encounters with the viral strain can mean a higher percentage of infection. The prevalence of the viral mutation in samples over the course of the study could also be due to sporadic outbreaks, super-distribution events or even holiday trips, said the epidemiologist at the University of Bern and co-author Emma Hodcroft Timesa structural biologist who was not involved in the study said Times he considers this to be an important advance, but notes that the way in which the virus interacts with other organisms needs to be further investigated for the functionality of the mutation. “It’s hard to know what these substitutions do,” said Jason McLellan of the University of Texas. “It really needs to be followed up with some extra experimental data.” To confirm and truly understand the data, the report notes that scientists need to examine a larger set of coronavirus samples than are currently available. of each of the virus mutation lines is unconvincing. Some of the tribes are prominent across the Midwest, others across the coast. However, they are unique to the US and each generation is usually a common sight in their own sectors of the country. The study was published in MedRxiv on Sunday and has yet to be judged by the peer.